Thursday, September 20, 2012


HISTORY OF

K.A.P.VISVANATHAM GOVERNMENT MEDICAL COLLEGE

 AND MAHATMA GANDHI MEMORIAL GOVT. HOSPITAL

TRICHY.

fp.M.ng.tp];tehjk; muR kUj;Jtf;fy;Y}hp 

kw;Wk;
kfhj;kh fhe;jp epidT muR kUj;Jtkid> jpUr;rp.

( Look in Baamini Tamil Fonts )
Prof.Dr.M.A.ALEEM, M.D.DM., (NEURO)

FORMER VICE PRINCIPAL,

HOD AND PROFESSOR OF NEUROLOGY,

KAPV GOVERNMENT MEDICAL COLLEGE, MGM GOVERNMENT HOSPITAL,

TRICHY-620 017. TAMILNADU INDIA . CELL:94431-59940

ek; kUj;Jtf;fy;Y}hpf;F tpj;jpl;ltH n[ayypjh mtHfs;:
  1. 22.12.1967y; jpUr;rpapy; muR kUj;Jtf;fy;Y}hp Jtq;fglNtz;Lk; vd jw;Nghija jkpof Kjy;tH khz;GkpF nry;tp.n[ayypjh(Selvi.J.Jayalalitha) mtHfspd; fhNthp je;j fiyr;nry;tp vd;Dk; eld epfo;r;rp vk;.[p.uhkre;jpud; jiyikapy; Nu];NfhH]; ikjhdj;jpy; khtl;l nghJ eyepjpf;fhf elj;jg;gl;L &.33>00>000/- (&gha; Kg;gj;jp %d;W yl;rk;;) t#y; nra;ag;gl;lJ.
  2. 22.08.1968y; jpUr;rpapy; muR kUj;Jtf;fy;Y}hp Jtq;f Ntz;Lk; vd cWg;gpdH ek;kho;thhpd; jPHkhdk; jpUr;rpuhg;gs;sp efH kd;w $l;lj;jpy; epiwNtw;wg;gl;lJ.
  3. 1986y; muR ,aw;gpay; fy;Y}Hp Jtq;fg;gl;L ek; fy;Y}hp cld; cWg;G epWtdkhf nray;gl;LtUfpwJ.
  4. 1997 Mk; Mz;L jpUr;rpapy; kUj;Jtf;fy;Y}hp Jtq;Ftjw;fhd muR Miz ntspaplg;gl;L 1998 Mk; Mz;L vk;.gp.gp.v];.> ,sepiy gbg;G khztH NrHiffSld; jpUr;rp muR kUj;Jtf; fy;Y}hp Jtq;fg;gl;lJ.
  5. 1997k; Mz;L kUj;Jtf;fy;tp gapy cjTk; tifapy; jpUr;rp mz;zy; fhe;jp epidT muR kUj;Jtkid (Annal Ganthi Memorial Govt Hospital) ek; kUj;Jt fy;Y}hpAld; ,izf;fg;gl;lJ.
  6. 26.06.2000 md;W jpUr;rp fp.M.ng.tp];tehjk; muR kUj;Jtf;fy;Y}hp vdg;ngahplg;gl;L mg;Nghija jkpofk; Kjy;tuhy; kUj;Jtf; fy;Y}hpf;fhd fl;blq;fSld;; jpwe;J itf;fg;gl;lJ.
  7. 10.03.2003 md;W ek; kUj;Jtf; fy;Y}hpf;fhd kpfg; nghpa fiyauq;fk; mg;Nghija Kjy;tH nry;tp.n[.n[ayypjh mtHfshy; jpwe;J itf;fg;gl;lJ.
  8. 2005k; Mz;L Kjy; b.vk;.vy;.b (DMLT) vd;Dk; kUj;Jt ,uj;jg; ghpNrhjid gl;lg;gbg;Gk;> b.MH.b.b (DDT, DRTT) Mfpa fjphpaf;fg; gl;lag;gbg;Gfs; Jtf;fp itf;fg;gl;ld.
  9. 2005k; Mz;L mtrurpfpr;ir njhopy;El;gH> Rthr rpfpr;ir njhopy; El;gH> ,uj;j Rj;jpfhpg;G njhopy;El;gH> kaf;ftpay; njhopy;El;gH> mWit rpfpr;ir muq;f njhopy;El;gH> KlePf;fpay; njhopy;El;gH> fjphpaf;f cjtpahsH Nghd;w kUj;Jt rhd;wpjo; kUj;Jtk; rhHe;j gbg;Gfs; ek; kUj;Jtf; fy;Y}hpapy; Jtq;fg;gl;ld.
  10. 28.01.2007y; fp.M.ng.tp];tehjk; kUj;Jt fy;Y}hpf;fhd 100 khztHfs; kw;Wk; 150 khztpaHfs; jq;Fk; tpLjp mg;Nghija Kjy;tuhy; jpwe;J itf;fg;gl;lJ.
  11. ,sepiy fy;Y}hpahf Jtf;fg;gl;l ek; kUj;Jt fy;Y}hp Muk;gpf;fg;gl;l 10 Mz;Lfspy; 2007y; KJepiy gbg;GfSld; $ba KJepiy kUj;Jtf;fy;Y}hpahf cUntLj;jJ.
  12. 2007k; Mz;L Kjy; vk;.b. nghJkUj;Jtk;> vk;.b.kfg;NgW kUj;Jtk; kw;Wk; vk;.v];. nghJ mWit rpfpr;ir Mfpa KJepiyg; gbg;Gfs; Jtf;fg;gl;ld.
  13. 18.02.2008y; KO msT %is euk;gpay; Jiw Vw;gLj;jp murhiz ntspaplg;gl;lJ.
  14. 2009k; Mz;L vk;.b.kaf;ftpay;> vk;.b.Foe;ijfs; ey kUj;Jt KJepiyg;gbg;Gfs; ek; fy;Y}hpapy; Muk;gpf;fg;gl;ld.
  15. 2011y; ek; fy;Y}hpapy; NjHT muq;fk; fl;Ltjw;Fk;> fy;Y}hp Kjy;tH jq;F tshfk; fl;Ltjw;Fk;> &.3.86 Nfhb xJf;fp murhiz khz;GkpF jkpof Kjy;tH n[.n[ayypjh ntspapl;Ls;shH.
ek; kUj;Jt kidia tpz;zsT caHj;jpatH n[ayypjh mtHfs;:
  1. ek; khtl;l kUj;Jt kid 1857y; fhe;jp khHnfl; gFjpapy; Jtq;fg;gl;L 152 Mz;LfSf;F Nkyhf ,aq;fp tUk; xU rhpj;jpuk; tha;e;j kUj;JtkidahFk;.
  2. 15.07.1950y; jw;NghJ cs;s kUj;Jtkid tshfj;jpy; fhe;jp[p epidT khtl;l jiyik kUj;Jtkid vd;w ngahpy; 24Vf;fH epyg;gug;gpy; mg;Nghija Kjy;tH gp.v];.Fkhurhkpuh[h vd;gtuhy; Jtq;fg;gl;lJ.
  3. 02.10.1969y; kfhj;kh fhe;jp E}w;whz;L epidT cs;Nehahspfs; gphpT fl;blk; ek; kUj;Jtkidapy; Jtq;fp itf;fg;gl;lJ.
  4. 21.06.1960y; nrd;id murhq;f Kjy;tuhy; fhe;jp epidT khtl;l kUj;Jtkidf;fhd GwNehahsp rprpr;ir epiyak; mbf;fy; ehl;lg;gl;L 28.01.1961y; nrd;id murhq;f Rfhju mikr;ruhy; jpwe;J itf;fg;gl;lJ.
  5. 1966y; vf;];Nu ek; kUj;Jtkidapy; njhlq;fg;gl;lJ.
  6. 31.12.1967y; Foe;ijfs; gphpT fl;blk; Jtq;fg;gl;L EVR kzpak;ik vd;w ngahpy; mg;Nghija jkpof Kjy;tH rp.vd;.mz;zhj;Jiw mtHfshy; jpwe;J itf;fg;gl;lJ.
  7. 1973y; jdp rpj;jh gphpT ek; kUj;Jtkid tshfj;jpy; Jtq;fg;gl;lJ.
  8. 10.03.1974 ek; kUj;Jtkidapy; Jiz nrtpypaH kfg;NgW cjtpahsH tpLjp mg;Nghija jkpof murpd; kf;fs; eytho;T mikr;ruhy; jpwe;J itf;fg;gl;lJ.
  9. 12.01.1976y; mg;Nghija jkpof MSeH Nkhfd;yhy; Rfhjpah ,k;kUj;Jtkidapy; mtru rpf;irg;gphpit (ICU) jpwe;J itj;jhH.
  10. 05.01.1977y; muR uh[h[p fhrNeha; kUj;Jtkid ek; kUj;JtkidAld; ,izf;fg;gl;lJ.
  11. 04.11.1977y; gpd; gpurt NgWfhy kw;Wk; mWitrpfpr;ir fl;blk; mg;Nghija jkpof murpd; r%f eyj;Jiw mikr;ruhy; jpwe;Jitf;fg;gl;lJ.
  12. 1979y; ek; kUj;Jtkidapy; ,uj;j tq;fp njhlq;fp itf;fg;gl;lJ.
  13. 1980y; [dthp khjk; ek; kUj;Jtkid mz;zy;fhe;jp epidT muR kUj;Jtkid vd kJiu vH];fpd; kUj;Jtkid ngaHkhw;wj;NjhL khw;wg;gl;ljhf $wg;gLfpwJ.
  14. 1980y; ek; kUj;Jtkidapy; nrtpypag;gs;sp Jtq;fg;gl;lJ.
  15. 28.04.1985y; nrtpypaH gapw;rpg;gs;sp tpLjp mg;Nghija jkpof eytho;T Jiw mikr;ruhy; ,k;kUj;Jtkidapy; Jtq;fg;gl;lJ.
  16. 1990y; vr;.I.tp. ghpNrhjid $lk; ek; kUj;Jtkidapy; epWtg;gl;lJ.
  17. 1997k; Mz;L ek; mz;zy;fhe;jp muR kUj;Jtkid fp.M.ng.tp];tehjk; muR kUj;Jtfy;Y}hpAld; ,izf;fg;gl;L kUj;Jtk; gapy;tpf;Fk; kUj;Jtkidahf juk; caHj;jg;gl;lJ.
  18. 26.10.1997y; fz; rpfpr;irg; gphptpw;F fl;blk; mg;Nghija Kjy;tuhy; mbf;fy; ehl;lg;gl;lJ.
  19. 28.03.1997y; rp.b.];Nfd; fUtp ek; kUj;Jtkidapy; epWtg;gl;L mg;Nghija jkpof murpd; czT mikr;ruhy; jpwe;J itf;fg;gl;lJ.
  20. 02.08.1999y; ,k;kUj;Jt kidapy; Foe;ijfs; njhl;by; gFjp mg;Nghija jpUr;rp khtl;l Ml;rpj;jiytH mtHfshy; jpwe;J itf;fg;gl;lJ.
  21. 29.09.2007y; vk;.MH.I. ];Nfd; ek; kUj;Jtkidapy; mg;Nghija jkpof Kjy;tuhy; Jtq;fp itf;fg;gl;lJ.
  22. 17.04.2010 KJepiy kw;Wk; gapw;rp kUj;Jt khztH jq;Fk; tpLjp mg;Nghija jkpof Rfhjhu mikr;ruhy; jpwe;J itf;fg;gl;lJ.
  23. 08.09.2010y; kdey cs;Nehahspfs; rpfpr;ir gphpT mg;Nghija jkpof Kjy;tuhy; jpwe;J itf;fg;gl;lJ.
  24. 25.01.2011y; jilapy;yh kpd;rhuk; toq;fg;gl;L mg;Nghija jkpof muR Nghf;Ftuj;J Jiw mikr;ruhy; jpwe;J itf;fg;gl;lJ.
  25. 24.01.2011y; kfspH kw;Wk; Foe;ijfs; gphpTf;fhf 6.26 Nfhb nrytpy; fl;blk; fl;l murhiz ntspaplg;gl;L fl;blg; gzpfs; ele;J tUfpd;wd.
  26. 24.01.2012y; ek; khz;GkpF jkpof Kjy;tH n[.n[ayypjh mtHfs; 2011 2012 muR epjpepiy mwpf;ifapy; jpUr;rp khtl;l gFjp kf;fspd; ePz;l ehs; Nfhhpf;ifahf gy;NtW njhz;L epWtdq;fs;> murpay;fl;rp gpuKfHfs;> cs;shl;rp cWg;gpdHfs; kw;Wk; ,g;gFjp kf;fspd; Nfhhpf;ifia fUj;jpy; nfhz;L kj;jpa muR juhky; 2003k; Mz;L Kjy; fplg;gpy; itf;fg;gl;bUe;j #g;gH ];ng]hypl;b (Super Speciality Hospital) kUj;Jtkid tshfj;jpy; njhlq;Ftjw;fhf &.100 Nfhbia mwptpj;J mjw;fhf fl;blk; epWt murhiz ntspapl;L mg;gzpfs; ele;J tUfpd;wd.
  27. 31.07.2012y; ek; mz;zy;fhe;jp muR kUj;Jtkid kfhj;kh fhe;jp epidT muR kUj;Jtkid vd ngaHkhw;wk; nra;J murhiz ntspaplg;gl;Ls;sJ.
,t;thW jpUr;rp muR kUj;Jtf;fy;Y}hp nfhz;L tuNtz;Lnkd 23.12.1967y; jkJ fhNthp je;j fiyr;nry;tp vd;Dk; ehlf epfo;r;rp %yk; tpj;jpl;L jw;NghJ tiu kUj;Jt fy;Y}hp kw;Wk; kUj;Jtkid Jtq;fg;gl;ljpy; ,Ue;J ngwg;glhj epjpahf 100 Nfhb &ghia ek; kUj;Jtkidia #g;gH ];ngrhypl;b kUj;Jtkidahf tpz;zsT caHj;jpl;l ek; khz;GkpF jkpof Kjy;tH n[.n[ayypjh mtHfSf;F vdJ ed;wpapidAk; ,f;fy;Y}hpapd; Kjy;tH> MrphpaHfs;> khztHfs; kw;Wk; ,g;gFjp kf;fs;> murpay; gpuKfHfs; kw;Wk; cs;shl;rp gpujpepjpfs; rhHghfTk; ed;wpapid njhptpj;Jf;nfhs;fpNwd;.
,f;fl;Liu cUthf Mjhuq;fs; nfhLj;J cjtpa jpUr;rp gj;jphpf;ifahsH fofk; (Press Club) nrayhsH jpU.e.nre;jpy;Nty; mtHfSf;Fk;> jpUr;rp `pe;J ehspjo; %j;jg;gj;jphpf;ifahsH fNzrd; mtHfSf;Fk; ed;wpapid njhptpj;Jf;nfhs;fpNwd;.

NguhrpaH lhf;lH.vk;.V.myPk; M.D.DM.(Neuro)
Kd;dhy; Jiz Kjy;tH kw;Wk; %is euk;gpay; Jiw NguhrpaH>
fp.M.ng.tp];tehjk; muR kUj;Jtf;fy;Y}hp kw;Wk;
kfhj;khfhe;jp epidT muR kUj;Jtkid>
jpUr;rp.



Tuesday, September 18, 2012


World Alzheimer’s Day:September 21-2012
Dementia-Living Together
Prof. Dr.M.A.Aleem.M.D.D.M.,(Neuro) HOD and professor of Neurology KAPV Government Medical college. MGM Government Hospital Trichy -620017.
Introduction:
Dementia is syndrome, usually of a chronic nature, caused by a variety of brain illnesses that affects memory, thinking, behaviour and ability to perform everyday activities. . Alzheimer,s disease(AD) is the most common cause of dementia and possibly contributes to up to 70% of cases. Alzheimer,s is a degenerative disease of the nervous system where the patient starts to lose their memory and in later cases even show behavioral disorders and forgets their own identity. Dementia is the major symptom of Alzheimer’s disease where the person loses the activity of the brain and the memory. The factors contributing to the disease are unknown; however 5 percent of the Alzheimer’s has been reported to have a hereditary origin. Though a number of researches are being done to find more on the disease and its cure, till now only symptomatic treatment are available for it and neither the reason behind the disease or its progression is clearly understood. The ignorance about the disease and its symptoms has wrecked a number of families and has turned their world upside down when they have to deal with a patient from their family. And the knowledge about what the disease actually is and how the patients should be taken care of can change the way of life of such caregivers and also patient who needs the most care and support in this difficult time. The ADI (Alzheimer’s Disease International) launched the International Alzheimer’s Day on 21st September 1994 on their tenth annual conference. Since then International Alzheimer’s Day or the World Alzheimer’s Day is observed every year on the 21st of September to spread the understanding of this degenerating disease of the central nervous system. The Alzheimer’s organization around the globe keeps their focus on the day by creating awareness about this progressive and irreversible disease which has million victm every part of the world every three minutes . . Alzheimer,s disease is increasing its number of patients every minute and so does it calls for the right education and guidance about it and its initial symptoms which we often never notice. Alzheimer’s disease is the major cause of dementia around the world and it is estimated that every year millions of lives are affected by it. Almost ten percent of adults above the age of 65 are affected by Alzheimer,s and it is believed that the incidence and the rate of disease will increase with age and time. There are also cases of early onset of the disease where people of 30 to 45 . Every year on the 21st of September, Alzheimer associations across the globe unite to recognize World Alzheimer’s Day. Together, we are making small but important strides toward increasing awareness and combating the stigma. Alzheiimer Disease International, the ‘global voice on dementia’, has given the theme of “Dementia – Living Together” to this year’s World Alzheimer Day2012.
Magnitude of problem:
Every 4 seconds, a New Case of Dementia in the World
Dementia and Alzheimer’s a Global Health Priority
Every 71 seconds, someone develops Alzheimer’s disease. Today, it is estimated that about 30 million people worldwide suffer from Alzheimer’s disease. Alzheimer’s affects about 10 percent of people aged 65 and up, and the prevalence doubles roughly every 10 years after age 65. Half of the population ages 85 and up may have Alzheimer’s. Because the population of the india is aging, the number of people with Alzheimer’s will continue to rise unless something can be done to stem the disease.
By 2050, there is expected to be one new case of AD every 33 seconds, or nearly million new cases per year, and AD prevalence is projected tobe 11 million to 16 million. Dramatic increases in the number of “oldest –old (those age> 85 years) across all racial and ethnic groups are expected to contribute to the increased prevalence of AD. Worldwide nearly 35.6 milion people live with dementia. This number is expected to double by 2030 (65.7 million) and more than triple by 2050 (115.4). Dementia affects people in all countries, with more than half (58%) living in low –and middle-income countries. By 2050, this is likely to rise to more than 70%. WHO recognises the size and complexity of the dementia challenge and countries to viwe dementia as a critical public heaith priority.
Dementia represents not only a public health crisis but a social and fiscal nightmare as well Around the world a new case of dementia arises every four seconds. That’s a staggering growth rate, equivalent to 7.7.million new cases of dementia every year –the same size as the populations of Switzerland and isreal. Our current health systems simply cannot cope with the explosion of the dementia crisis as we all live longer ; this is as much an economic and fiscal disaster waiting to happen as it is a social and health challenge of the highest order. Alzheimer’s disease in particular-as a “ticking time bomb” given the rapid growth in aging populations worldwide. There would be 35 million people worldwide with dementia by 2010. That number is set to almost double every 20 years to 65.7 million in 2030 and 115.4 million in 2050.
In India Scientists had earlier said that by 2020,around 10 million indians above the age of 65 would suffer from dementia. By 2040, the number would incerase to around 22 million. Going by the new estimaties, the report said the percentage increase of the number of peopel with dementia in the next 20 years will stand at 107% in south Aisa, 134% in latin America and 125% in north America and Middle East. A few, like india ,have national strategies developed by civil society organisations, and we hope this report will prompt their governments to adapt these into official national plans . since we know the prevalence of the disease will explode in this century as we all live longer-the risk of dementia is 1 in 8 for those over 65 and a shocking 1 in 2.5 for those over 85-its impact will become greater as the decades go by .
The report recommended that the WHO declare dementia a health priority,and that countries including india develop a plan for dealing with the greater numbers of dementia patients. The study had earliar made an interesting finting. When compared to China and Latin America, indian relatives were much less likely to acknowledge that the elders in their households were suffering from memory loss.
Alzheimer’s disease clinical stages.
The Leading Theory :
Alzheimer’s disease process beings when deposits of betaamyloid protein collect out side neurons (brain cells), gradually destroing synapses (the connection that enable neurons to receive and communicate information). The next step in the process occurs when neurofibrillary tangles made of tau protein accumlate inside neurons, interfering with normal cell function and eventually killing them. As neurons die and synapses wither, brain tissue shrings in key areas that support memory, language, and other thinking abilities in Alzhimer’s dementia.
The Unnoticed Fact:
About 30% of older people have amyloid deposits that might suggest Alzheimer’s later, others are spared.
It is possible to predict, on an individual basis, which people with early Alzheimer’s pathology will go on to develop the sevear memory and thinking problems that characterize the disease?
By better delineating the three stages of Alzheimer’s disease progression, researchers hope one day to find a way to intervene in the diseaes process in the people most at risk.
Stage 1:preclinical:
In this stage the patiens are mostly asymptomatic but pathological changes are properly already underway in the brain.
The section of the new guidelines that discusses the preclinical stage of Alzheimer’s does not provide diagnostic advice since there is nothing to diagnose but instead describes how researchers might use five biomarkers to estimate the changes of whether someone mihgt be at the preclinical stage. of Alzheimer’s, but also to learn what facter might predict a transition to mild cognitive impairment.
Stage 2: Mild Cognitive Impairment:
Mild cognitive impairment is an intermediate state between the normal forgetfulness that and thinking deficits that occur in Alzheimer’s. within five year, about half the people with mild cognitive impairment develop dementia most often Alzheimer’s As many as one quarter of those with mild cognitive impairment regain normal cognitive functioning in time. The rest remain stable. The new guidelines not only outline criteria whether someone has mild cognitive impairment, but also describe how biomarker tests might be used to predict whether they are likely to progress to Alzheimer’s.The foundation of diagnosis is a through evaluation to rule out other medical problems that might cause problems in thinking, such as heart disease, stroke, depression, or head trama. There are no standart neuropsychological test to diagnose mild cognitive imparment, so clinicians adapt those developed for other purposes. Because other thinking areas may deteriorate in parallel with or even before memory, the guidelines also recommend testing a varity of cognitive function. The following pattern of findings may suggest mild cognitive impairment: 1)Change In Cognition: A deterioration in thinking ability noticed either by the person affected, a loved one, or a clinician. 2)Impairment of one or more abilities: Including memory, attention, language, and ability to plan Ability To Function Independently: Some clinicians may suggest biomarker testing in a person with mild cognitive impairment if the results could guide treatment or help with life planning. Although some people in this position might not want to know what the future holds, others do-to allow time for making medical and financial plans or enabling participation in research studies. Stage 3:Dementia Due To Alzheimr’s Disease:
Once Alzheimer’s has progreassed to the point where memoey, thinking, and behavior are so impaired that a person is no longer able to function independently, the diagnosis is dementia.Diagnosis of Alzheimer’s dementia relies primarily on clinical signs and symptoms, along with tests to rule out other types of dementia or neurological illnesses.The changes in thinking and confirmed by a knowledgeable obsever, such as a spouse or close friend.Further testing may be necessary to confirm The nature and extent of cognitive impairment. The new criteria emphasize that memory impairment althought the most common initial symptom may not be the only one. After stroke, delirium, and other possible causes of dementia are ruled out, a diagnosis of Alzheimer’s is probable when cognitive or behavioral impairment develops gradualls, increased over time, and involves at least two of the following cognitive domans. Memory: This is the most common problem area and typically involves episodic memory(difficulty learning or remembering new information). In day-to-day life this might cause someone to misplace personal belongings, repeat the same question or conversation, forget things or get lost while walking or driving in familiar areas. Executive Function : This type of thinking indiudes planning, reasoning, judgment, and problem solving. Impairmend of executive function might manifest as difficulty with finances, failure to appreciate safety risks, or inapility to organize meals. Visuospatial albility: This refer to the ability to interpret visual information and see how objects fit into surroundings. Impairments in visuospatial ability may manifest in many different ways, such as trouble recognizing familiar people, or the inability to find objects such as eating utensils (even when in plain view). Language: Impairments in this domain might show up as hesitation in speaking, problems coming up with the right word, or spelling errors. Behaviour and personality: Uncharacteristic changes in behavior and personality include agitation, apathy, mood swings, obsessive or compulsive behavior, or socially unacceptable behavior. Conclusion:
Awareness about this degenerative and irreversible disorder is very important to understand and care for the patients affected with Alzheimer’s. The World Alzheimer’s Day focuses on getting more people know about the disease and why and how the patients could be managed though the disease itself cannot be cured or controlled as per the developement now. The first people who should be educated on the symptoms and management of the disease are those who have someone suffering from it.
In most of the cases the early Alzheimer’s symptoms are mistaken for age related forgetfulness and the disease is diagnosed only after it advanceds to higher stage making it equally difficult for the patient and for the caregivers. Being patient and knowing the symptoms and the extent to which the disease can proress will help in taking care of the patient and bearing with the pain of seeing their loved ones deteriorate into a living log.
The International Alzheimer’s day tells the people that they are not alone in their misery and someone next to them are facing the same or similar trauma, the talk show and the articles on the disease helps in educating the world t o know more about the disease. The day also promotes the donation into the fund for Alzheimer’s research which may someday provide a breakthrough for the treatment and new findings into key to unlock the disease.
Only eight countries worldwide currently have national programmes in place to address dementia. A new report Dementia: a public health priority, published by the World Health Organization (WHO) and Alzheimer’s Disease International, recommends that programmes focus on improving early diagnosis; raising public awareness about the disease and reducing stigma; and providing better care and more support to caregivers


Improving early diagnosis:
Lack of diagnosis is a major problem. Even in high-income countries, only one fifth to one half of cases of dementia are routinely recognized. When a diagnosis is made, it often comes at a relatively late stage of the disease.“We need to increase our capacity to detect dementia early and to provide the necessary health and social care.“Health-care workers are often not adequately trained to recognize dementia.”
There is a general lack of information and understanding about dementia. This fuels stigma, which in turn contributes to the social isolation of both the person with dementia and their caregivers, and can lead to delays in seeking diagnosis, health assistance and social support.
“Public awarness about dementia, its symptoms, the importance of getting a diagnosis, and the help available for those with the condition is very limited. It is now vital to tackle the poor levels of public awareness and understanding, and to drastically reduce the stigma associated with dementia.”
Strengthening care is also a key. In every region of the world, most caregiving is provided by informal caregivers – spouses, adult children, other family members and friends. The report notes that people who care for a person with dementia are themselves particularly prone to mental disorders, such as depression and anxiety, and are often in poor physical health themselves. Many caregivers also suffer economically as they may be forced to stop working, cut back on work, or take a less demanding job to care for a family member with dementia.
‘Alzheimer’s, among all non-communicable diseases, demends our urgent and serious focus.Alzheimer’s will surely become the fiscal nightmore of the 21 st centery, quite apart from the personal, family and community devastation it causes in its wake. By working togather to address these commen risk facter’s the global health community can make great inroads into the burten of disese caused by Alzheimer’s disese and these other NCDs(Non-communicable diseases), that together cause 3 in 5 of all deaths. Dementia is not a normal part of aging. 35.6 million people were estimated to be living with demendia in 2010. There are 7.7 milion new cases of demendia each year, implaying that there is a new case of demendia somewhere in the world every four seconds. The accelerating rates of dementia are cause for immediate action, especially in low- and middle- income countries where resources are few. People live for many years after the onset of symptoms of dementia. With apporiate support, many can and should be enabled to continue to engage and contribute within society and have a good quality of life.Dementia is overwhelming for the caregivers and adequate support is required for them from the health, social, financial and legal systems.





HISTORY OF
K.A.P.VISVANATHAM GOVERNMENT MEDICAL COLLEGE AND MAHATMA GANDHI MEMORIAL GOVT. HOSPITAL TRICHY.

fp.M.ng.tp];tehjk; muR kUj;Jtf;fy;Y}hp kw;Wk; kfhj;kh fhe;jp epidT muR kUj;Jtkid> jpUr;rp.

Prof.Dr.M.A.ALEEM, M.D.DM., (NEURO)
FORMER VICE PRINCIPAL,
HOD AND PROFESSOR OF NEUROLOGY,
KAPV GOVERNMENT MEDICAL COLLEGE, MGM GOVERNMENT HOSPITAL,
TRICHY-620 017. TAMILNADU INDIA . CELL:94431-59940

ek; kUj;Jtf;fy;Y}hpf;F tpj;jpl;ltH n[ayypjh mtHfs;:
  1. 22.12.1967y; jpUr;rpapy; muR kUj;Jtf;fy;Y}hp Jtq;fglNtz;Lk; vd jw;Nghija jkpof Kjy;tH khz;GkpF nry;tp.n[ayypjh(Selvi.J.Jayalalitha) mtHfspd; fhNthp je;j fiyr;nry;tp vd;Dk; eld epfo;r;rp vk;.[p.uhkre;jpud; jiyikapy; Nu];NfhH]; ikjhdj;jpy; khtl;l nghJ eyepjpf;fhf elj;jg;gl;L &.33>00>000/- (&gha; Kg;gj;jp %d;W yl;rk;;) t#y; nra;ag;gl;lJ.
  2. 22.08.1968y; jpUr;rpapy; muR kUj;Jtf;fy;Y}hp Jtq;f Ntz;Lk; vd cWg;gpdH ek;kho;thhpd; jPHkhdk; jpUr;rpuhg;gs;sp efH kd;w $l;lj;jpy; epiwNtw;wg;gl;lJ.
  3. 1986y; muR ,aw;gpay; fy;Y}Hp Jtq;fg;gl;L ek; fy;Y}hp cld; cWg;G epWtdkhf nray;gl;LtUfpwJ.
  4. 1997 Mk; Mz;L jpUr;rpapy; kUj;Jtf;fy;Y}hp Jtq;Ftjw;fhd muR Miz ntspaplg;gl;L 1998 Mk; Mz;L vk;.gp.gp.v];.> ,sepiy gbg;G khztH NrHiffSld; jpUr;rp muR kUj;Jtf; fy;Y}hp Jtq;fg;gl;lJ.
  5. 1997k; Mz;L kUj;Jtf;fy;tp gapy cjTk; tifapy; jpUr;rp mz;zy; fhe;jp epidT muR kUj;Jtkid (Annal Ganthi Memorial Govt Hospital) ek; kUj;Jt fy;Y}hpAld; ,izf;fg;gl;lJ.
  6. 26.06.2000 md;W jpUr;rp fp.M.ng.tp];tehjk; muR kUj;Jtf;fy;Y}hp vdg;ngahplg;gl;L mg;Nghija jkpofk; Kjy;tuhy; kUj;Jtf; fy;Y}hpf;fhd fl;blq;fSld;; jpwe;J itf;fg;gl;lJ.
  7. 10.03.2003 md;W ek; kUj;Jtf; fy;Y}hpf;fhd kpfg; nghpa fiyauq;fk; mg;Nghija Kjy;tH nry;tp.n[.n[ayypjh mtHfshy; jpwe;J itf;fg;gl;lJ.
  8. 2005k; Mz;L Kjy; b.vk;.vy;.b (DMLT) vd;Dk; kUj;Jt ,uj;jg; ghpNrhjid gl;lg;gbg;Gk;> b.MH.b.b (DDT, DRTT) Mfpa fjphpaf;fg; gl;lag;gbg;Gfs; Jtf;fp itf;fg;gl;ld.
  9. 2005k; Mz;L mtrurpfpr;ir njhopy;El;gH> Rthr rpfpr;ir njhopy; El;gH> ,uj;j Rj;jpfhpg;G njhopy;El;gH> kaf;ftpay; njhopy;El;gH> mWit rpfpr;ir muq;f njhopy;El;gH> KlePf;fpay; njhopy;El;gH> fjphpaf;f cjtpahsH Nghd;w kUj;Jt rhd;wpjo; kUj;Jtk; rhHe;j gbg;Gfs; ek; kUj;Jtf; fy;Y}hpapy; Jtq;fg;gl;ld.
  10. 28.01.2007y; fp.M.ng.tp];tehjk; kUj;Jt fy;Y}hpf;fhd 100 khztHfs; kw;Wk; 150 khztpaHfs; jq;Fk; tpLjp mg;Nghija Kjy;tuhy; jpwe;J itf;fg;gl;lJ.
  11. ,sepiy fy;Y}hpahf Jtf;fg;gl;l ek; kUj;Jt fy;Y}hp Muk;gpf;fg;gl;l 10 Mz;Lfspy; 2007y; KJepiy gbg;GfSld; $ba KJepiy kUj;Jtf;fy;Y}hpahf cUntLj;jJ.
  12. 2007k; Mz;L Kjy; vk;.b. nghJkUj;Jtk;> vk;.b.kfg;NgW kUj;Jtk; kw;Wk; vk;.v];. nghJ mWit rpfpr;ir Mfpa KJepiyg; gbg;Gfs; Jtf;fg;gl;ld.
  13. 18.02.2008y; KO msT %is euk;gpay; Jiw Vw;gLj;jp murhiz ntspaplg;gl;lJ.
  14. 2009k; Mz;L vk;.b.kaf;ftpay;> vk;.b.Foe;ijfs; ey kUj;Jt KJepiyg;gbg;Gfs; ek; fy;Y}hpapy; Muk;gpf;fg;gl;ld.
  15. 2011y; ek; fy;Y}hpapy; NjHT muq;fk; fl;Ltjw;Fk;> fy;Y}hp Kjy;tH jq;F tshfk; fl;Ltjw;Fk;> &.3.86 Nfhb xJf;fp murhiz khz;GkpF jkpof Kjy;tH n[.n[ayypjh ntspapl;Ls;shH.
ek; kUj;Jt kidia tpz;zsT caHj;jpatH n[ayypjh mtHfs;:
  1. ek; khtl;l kUj;Jt kid 1857y; fhe;jp khHnfl; gFjpapy; Jtq;fg;gl;L 152 Mz;LfSf;F Nkyhf ,aq;fp tUk; xU rhpj;jpuk; tha;e;j kUj;JtkidahFk;.
  2. 15.07.1950y; jw;NghJ cs;s kUj;Jtkid tshfj;jpy; fhe;jp[p epidT khtl;l jiyik kUj;Jtkid vd;w ngahpy; 24Vf;fH epyg;gug;gpy; mg;Nghija Kjy;tH gp.v];.Fkhurhkpuh[h vd;gtuhy; Jtq;fg;gl;lJ.
  3. 02.10.1969y; kfhj;kh fhe;jp E}w;whz;L epidT cs;Nehahspfs; gphpT fl;blk; ek; kUj;Jtkidapy; Jtq;fp itf;fg;gl;lJ.
  4. 21.06.1960y; nrd;id murhq;f Kjy;tuhy; fhe;jp epidT khtl;l kUj;Jtkidf;fhd GwNehahsp rprpr;ir epiyak; mbf;fy; ehl;lg;gl;L 28.01.1961y; nrd;id murhq;f Rfhju mikr;ruhy; jpwe;J itf;fg;gl;lJ.
  5. 1966y; vf;];Nu ek; kUj;Jtkidapy; njhlq;fg;gl;lJ.
  6. 31.12.1967y; Foe;ijfs; gphpT fl;blk; Jtq;fg;gl;L EVR kzpak;ik vd;w ngahpy; mg;Nghija jkpof Kjy;tH rp.vd;.mz;zhj;Jiw mtHfshy; jpwe;J itf;fg;gl;lJ.
  7. 1973y; jdp rpj;jh gphpT ek; kUj;Jtkid tshfj;jpy; Jtq;fg;gl;lJ.
  8. 10.03.1974 ek; kUj;Jtkidapy; Jiz nrtpypaH kfg;NgW cjtpahsH tpLjp mg;Nghija jkpof murpd; kf;fs; eytho;T mikr;ruhy; jpwe;J itf;fg;gl;lJ.
  9. 12.01.1976y; mg;Nghija jkpof MSeH Nkhfd;yhy; Rfhjpah ,k;kUj;Jtkidapy; mtru rpf;irg;gphpit (ICU) jpwe;J itj;jhH.
  10. 05.01.1977y; muR uh[h[p fhrNeha; kUj;Jtkid ek; kUj;JtkidAld; ,izf;fg;gl;lJ.
  11. 04.11.1977y; gpd; gpurt NgWfhy kw;Wk; mWitrpfpr;ir fl;blk; mg;Nghija jkpof murpd; r%f eyj;Jiw mikr;ruhy; jpwe;Jitf;fg;gl;lJ.
  12. 1979y; ek; kUj;Jtkidapy; ,uj;j tq;fp njhlq;fp itf;fg;gl;lJ.
  13. 1980y; [dthp khjk; ek; kUj;Jtkid mz;zy;fhe;jp epidT muR kUj;Jtkid vd kJiu vH];fpd; kUj;Jtkid ngaHkhw;wj;NjhL khw;wg;gl;ljhf $wg;gLfpwJ.
  14. 1980y; ek; kUj;Jtkidapy; nrtpypag;gs;sp Jtq;fg;gl;lJ.
  15. 28.04.1985y; nrtpypaH gapw;rpg;gs;sp tpLjp mg;Nghija jkpof eytho;T Jiw mikr;ruhy; ,k;kUj;Jtkidapy; Jtq;fg;gl;lJ.
  16. 1990y; vr;.I.tp. ghpNrhjid $lk; ek; kUj;Jtkidapy; epWtg;gl;lJ.
  17. 1997k; Mz;L ek; mz;zy;fhe;jp muR kUj;Jtkid fp.M.ng.tp];tehjk; muR kUj;Jtfy;Y}hpAld; ,izf;fg;gl;L kUj;Jtk; gapy;tpf;Fk; kUj;Jtkidahf juk; caHj;jg;gl;lJ.
  18. 26.10.1997y; fz; rpfpr;irg; gphptpw;F fl;blk; mg;Nghija Kjy;tuhy; mbf;fy; ehl;lg;gl;lJ.
  19. 28.03.1997y; rp.b.];Nfd; fUtp ek; kUj;Jtkidapy; epWtg;gl;L mg;Nghija jkpof murpd; czT mikr;ruhy; jpwe;J itf;fg;gl;lJ.
  20. 02.08.1999y; ,k;kUj;Jt kidapy; Foe;ijfs; njhl;by; gFjp mg;Nghija jpUr;rp khtl;l Ml;rpj;jiytH mtHfshy; jpwe;J itf;fg;gl;lJ.
  21. 29.09.2007y; vk;.MH.I. ];Nfd; ek; kUj;Jtkidapy; mg;Nghija jkpof Kjy;tuhy; Jtq;fp itf;fg;gl;lJ.
  22. 17.04.2010 KJepiy kw;Wk; gapw;rp kUj;Jt khztH jq;Fk; tpLjp mg;Nghija jkpof Rfhjhu mikr;ruhy; jpwe;J itf;fg;gl;lJ.
  23. 08.09.2010y; kdey cs;Nehahspfs; rpfpr;ir gphpT mg;Nghija jkpof Kjy;tuhy; jpwe;J itf;fg;gl;lJ.
  24. 25.01.2011y; jilapy;yh kpd;rhuk; toq;fg;gl;L mg;Nghija jkpof muR Nghf;Ftuj;J Jiw mikr;ruhy; jpwe;J itf;fg;gl;lJ.
  25. 24.01.2011y; kfspH kw;Wk; Foe;ijfs; gphpTf;fhf 6.26 Nfhb nrytpy; fl;blk; fl;l murhiz ntspaplg;gl;L fl;blg; gzpfs; ele;J tUfpd;wd.
  26. 24.01.2012y; ek; khz;GkpF jkpof Kjy;tH n[.n[ayypjh mtHfs; 2011 2012 muR epjpepiy mwpf;ifapy; jpUr;rp khtl;l gFjp kf;fspd; ePz;l ehs; Nfhhpf;ifahf gy;NtW njhz;L epWtdq;fs;> murpay;fl;rp gpuKfHfs;> cs;shl;rp cWg;gpdHfs; kw;Wk; ,g;gFjp kf;fspd; Nfhhpf;ifia fUj;jpy; nfhz;L kj;jpa muR juhky; 2003k; Mz;L Kjy; fplg;gpy; itf;fg;gl;bUe;j #g;gH ];ng]hypl;b (Super Speciality Hospital) kUj;Jtkid tshfj;jpy; njhlq;Ftjw;fhf &.100 Nfhbia mwptpj;J mjw;fhf fl;blk; epWt murhiz ntspapl;L mg;gzpfs; ele;J tUfpd;wd.
  27. 31.07.2012y; ek; mz;zy;fhe;jp muR kUj;Jtkid kfhj;kh fhe;jp epidT muR kUj;Jtkid vd ngaHkhw;wk; nra;J murhiz ntspaplg;gl;Ls;sJ.
,t;thW jpUr;rp muR kUj;Jtf;fy;Y}hp nfhz;L tuNtz;Lnkd 23.12.1967y; jkJ fhNthp je;j fiyr;nry;tp vd;Dk; ehlf epfo;r;rp %yk; tpj;jpl;L jw;NghJ tiu kUj;Jt fy;Y}hp kw;Wk; kUj;Jtkid Jtq;fg;gl;ljpy; ,Ue;J ngwg;glhj epjpahf 100 Nfhb &ghia ek; kUj;Jtkidia #g;gH ];ngrhypl;b kUj;Jtkidahf tpz;zsT caHj;jpl;l ek; khz;GkpF jkpof Kjy;tH n[.n[ayypjh mtHfSf;F vdJ ed;wpapidAk; ,f;fy;Y}hpapd; Kjy;tH> MrphpaHfs;> khztHfs; kw;Wk; ,g;gFjp kf;fs;> murpay; gpuKfHfs; kw;Wk; cs;shl;rp gpujpepjpfs; rhHghfTk; ed;wpapid njhptpj;Jf;nfhs;fpNwd;.
,f;fl;Liu cUthf Mjhuq;fs; nfhLj;J cjtpa jpUr;rp gj;jphpf;ifahsH fofk; (Press Club) nrayhsH jpU.e.nre;jpy;Nty; mtHfSf;Fk;> jpUr;rp `pe;J ehspjo; %j;jg;gj;jphpf;ifahsH fNzrd; mtHfSf;Fk; ed;wpapid njhptpj;Jf;nfhs;fpNwd;.

NguhrpaH lhf;lH.vk;.V.myPk; M.D.DM.(Neuro)
Kd;dhy; Jiz Kjy;tH kw;Wk; %is euk;gpay; Jiw NguhrpaH>
fp.M.ng.tp];tehjk; muR kUj;Jtf;fy;Y}hp kw;Wk;
kfhj;khfhe;jp epidT muR kUj;Jtkid>
jpUr;rp.



Saturday, September 15, 2012

1st Asia Pacific Stroke conference Tokyo 2012

Attended 1st Asia Pacific Stroke conference was held at Hilton hotel Tokyo on13th September 2012 by Ferer. its useful informative. Many aspect on stroke were discussed.

Thursday, September 6, 2012

SUICIDE – YOUNG AT RISK IN INDIA FOR World Suicide Prevention Day 10/09/2012


World Suicide Prevention Day 10/09/2012
SUICIDE – YOUNG AT RISK IN INDIA

Dr.M. A.ALEEM.M.D.D.M.,(Neuro)
Consultant Neurologist and Epileptologist.
Trained at Institution of neurology Queen’s Square London.
Professor of Neurology KAPV Government Medical college.
MGM Government Hospital Trichy -620017. Tamilnad,India.

 Introduction:
                          On average, almost 3000 people commit suicide daily. For every person who completes a suicide, 20 or more may attempt to end their lives. At the global level, awarness needs to be raised that suicide is a major preventable cause of premature death.Every year World Suicide Prevention Day on 10 September is adopted to promotes worldwide commitment and action to prevent Suicides September 10th, 2012 marks the 10th anniversary of the world Suicide Prevention Day: ten years of research, ten yars of prevention, ten years of education and dissemination of information.The theme of world Suicide Prevention Day this year 2012 is “Suicide Prevention across the Globe: Strengthening Protective Factors and Instilling Hope”
 The Magnitude of the problem:
                        Suicidal behaviour has become a major public health problem across the world. It is a complex phenomenon that usually occurs along a continuum, progressing from suicidal thoughts, to planning, to attempting suicide, and finally dying by suicide, which represents the final tragic outcome of a morbid process. Approximately one million people worldwide die by suicide each year. This corresponds to one death by suicide every 40 seconds.  Suicide attempts and suicidal ideation are far more common; for example, the number of suicide attempts may be up to 20 times the number of deaths by suicide. It is estimated that about 5% of persons attempt suicide at least once in their life and that the lifetime prevalence of suicidal ideation in the general population is between 10 and 14%. Suicide is one of the leading causes of death in the world and over the last years rates have increased by 60% in some countries. In addition, suicide statistics may not always be accurate. Many suicides are hidden among other causes of death, such as single car, single driver road traffic accidents, unwitnessed drownings and other undetermined deaths. In addition, suicide is estimated to be under-reported for multiple reasons including stigma, religious concerns and social attitudes. The psychological and social impact of suicide of the family and community is enormous.                                                                                                                                                                                                               Statistics:
<!--[if !supportLists]-->§  <!--[endif]-->Every year, almost one million people die from suicida;a ”global” mortality rate of 16 per 100,000, or one death every 40 seconds.

<!--[if !supportLists]-->§  <!--[endif]-->In the last 45 years suicide rates have increased by 60% worldwide. . Suicide is among the three leading causes of death among those aged 15-44 years in some countries, and the second leading cause of death in the 10-24 years age group;these figures do not include . suicide attempts which are up to 20 times more frequent than completed suicide.


<!--[if !supportLists]-->§  <!--[endif]-->Suicide worldwide is estimated to represent 1.8% of the total global burden of disease in 1998 and 2.4% in countries with market and former socialist economies in 2020.

<!--[if !supportLists]-->§  <!--[endif]--> Although traditionally suicide rates have been highest among the male elderly, rates among young people have been increasing to such an extent that they are now the group at highest risk in a third of countries, in both developed and developing countries.

<!--[if !supportLists]-->§  <!--[endif]-->Mental disorders (particularly depression and alcohol use disorders) are a major risk factor for suicide in Europe and North America; however, in Asian countries impulsiveness plays an important role. Suicide is complex with psychological, social, biological, cultural and environment factors involved.

Problems in India:
                        Suicide in India is slightly above world rate. Of the half million people reported to die of suicide worldwide every year, 20% are Indians, for 17% of world population. In the last two decades, the suicide rate has increased from 7.9 to 10.3 per 100,000 with very high rates in some southern regions. In a study published in The Lancet in June 2012, the estimated number of suicides in India in 2010 was about 187,000, making the cause of 3 percent of death that  year. A large proportion of adult suicide deaths were found to occur between the ages of 15 years and 29 years, especially in women. Suicide attempters are ten times the suicide completers. In India age standardized are suicide death rate per 100,000 people at all age 18.6for boys and 12.7 for girls and women. Suicide become the second leading cause of death among the young in India.                                                                                                                                                                                          
                        The WHO reports about 1 million suicides a year, which would be a rate of about 14 per 100,000 in a global population of 7 billion. Suicide may soon be leading cause of death in india. In our country among men, 40 per cent of suicides were among people age 15-29. For women, it was nearly 60 per cent.
                        The rapid changes on society that have come with globlalization, the breakdown of the families. Suicide become the second-leading cause of death among the young in India. “The young face very high competition and pressure from families to succeed. Many parents think their child should come first in the class. Of course, that can’t happen,” may also lead to suicide . When youths start to despair, they often don’t think to seek help, or shun the idea because “they think psychiatry is only for crazy people,” So most of them landed in suicide. The risk of completing a suicide was 43% higher in men, who finished secondary or higher education, in comparison to those who had not completed primary education. Among women, the risk increased to 90%.
                             Four o f India’s southern states Tamil nadu, Andhra Pradesh, Karnataka and kerala  that together constitute  22% of the country’s population recorded 42% of suicide deaths in men and 40% of self-inflicted fatalities in women in 2010.Maharashtra and West Bengal together accounted for an additional 15% of suicide deaths. Delhi recorded the lowest suicide rate in the country. In absolute numbers, the most suicide deaths in individuals, aged 15 years or older, were in AP (28,000), Tamil Nadu (24,000) and Maharashtra (19,000). Of the total deaths by suicide in individuals aged 15 years or older, about 40% suicide deaths in men and about 56% in women occurred in individuals aged 15-29 years. Suicides deaths occurred at younger ages in women (average age 25 years) than in men (average age 34 years). Educated persons were at greater risk of completing a suicide.
                                                About half of suicide deaths (49% among men, and 44% among women) were due to poisoning(36.6%), mainly ingesting of pesticides. Hanging was the second(32.1%) most common cause for men and women, while burns(7.9%) accounted for about one-sixth of suicides by women.
                        
Age differences:
                            Suicidal behaviour can occur at any age. The frequency of  suicidal behaviour escalates steeply from childhood through middle to late adolescence and into adulthood. Suicide ranks as the second cause of death worldwide among 15-19 year olds, with at least 100,000 adolescents dying by suicide every year. Suicide rates are high among middle-aged and older adults and highest among those aged 75 and older. Elderly people are likely to have higher suicidal intent and use more lethal methods than younger people, and they are less likely to survive the physical consequences of an attempt.
Gender Differences:
                        On average, there are about three male suicides for every female suicide. This is more or less consistent across different age groups and in almost every country in the world. Conversely, rates of suicide attempts tend to be 2-3 time higher in women than in men, although the gender gap has narrowed in recent years. The disparity in suicide rates has been partly explained by the use of more lethal means and the experience of more aggression and higher intent to die, when suicidal, in men than women.There was a 64% correlation between domestic violence of women and suicidal ideation.
 Who is at risk of Suicide?  
(i) People with a history of suicide attempts of self harm:
                           A history of previous suicide attempt(s) of self harm is the strongest predictor of future death by suicide, corresponding to a 30-40 times higher suicide rate than the general population. The first days and weeks following psychiatric hospitalization represent the most critical period of suicide risk for patients. This finding highlights the need to attend carefully to continuity of care for psychiatric patients.

(ii) People with a psychiatric disorder and/or substance-related disorder:
                            It has beendocumented that approximately half of the people who seriously consider taking their lives have been diagnosed with a mental disorder during their life, and that up to 90% of people who die by suicide have dt least one psychiatric diagnosis. Among all dianoses, depressive disorders are most commonly associated with suicidal behaviour, followed by substance-related disorders, schizophrenia and personality disorders. Alcohol and drug abuse and dependence have been identified as important risk factors for suicidal thoughts and behaviours. Current substance use, even in the absence of abuse or debendence, is a significant rick factor for unplanned suicide attempts among those with suicidal thoughts. Comorbidity,namely the presence of two or more psychiatric disorders or a psychiatric disorder and a substance use disorder, significantly increases the risk of suicide.
  (iii)Those who experience stressful life events:
                              Stressful life events often act as precipitants of suicide attempts or suicide by those with a diminished capacity to cope with them. Impulsive attempts may follow stressful life events, including family and interpersonal conflicts, relationship breakdowns, other interpersonal difficulties, the presence of legal/disciplinary problems, and financial and job difficulties. Periods of economic crisis and unemployment are associated with greater social vulnerability and often an increase in deaths by suicide.Bereavement, consistently described as one of life’s most stressful events, has been shown to elevate the risk of suicide and suicidal behaviour in vulnerable people, particularly if the death is by suicide. The rick of suicide is aiso greater among patients with severe physical illnesses, such as cancer or HIV infection – in face, increased suicide rick has been found to be associated with a large number of medical conditions, ranging from asthma to traumatic brain injury. The experience of persistent stress also may explain the elevated risk of suicide in some occupations, such as physicians, military personnel and police officers, as well among people in prison. Moreover distal stressors, e.g. childhood trauma, have consistently been linked to an increased risk of suicidal behaviour in adult life.
(iv) Young at risk in India                                   
                        Young Indians are more likely to commit suicide than previously thought, especially those living in weathier and more educated regions, according to recent India’s rapid development is driving many youths to despair. Opportunities that have come with two decades of economic boom and open markets have also brought more job anxiety, higher expectations and more pressure to achieve, mental health.
                        Suicide rates are highest in the 15-29 age group, peaking in southern regions that are considered richer and more developed with better education, social welfare and health care. That puts the young at high risk-a new phenomenon experts said has happened recently as more middle-class youths strive to meet achievement expectations, and new technologies like cell phones and social networking sites help break down traditional family units once relied on for support. Few likely reasons for the rise in suicide among young people beyond the increased pressure that has come with new economic opportunity and social fragmentation.  The higher rates may come from “the greater likelihood of disaponintments when aspirations that define success and happiness are distorted or unmet by the reality faced by young people in a rapidly changing society are also a few likely factors to commit suicide by young in India.
Strengthening Protective Factors and Instilling Hope
(i) Protective factors:
                          Despite the wide experience of the above-cited rick factors in populations, the fact that completed suicide is a relatively rare event indicates that there are a range of protective factors that act to mitigate the effects of exposure to risk factors. Among psychological factors, resilience (the ability to cope with adverse life events and adjust to them), a sense of personal self-worth and self-confidence, effective coping and problem-solving skills, and adaptive help-seeking behaviour are ofter considered to be protective against the development of suicidal behaviours. Social and cultural factors such as religious and social integration, social connectedness and maintenance of good relationships with friends, colleagues and neighbours, access to support from relevant others and ready access to health care are associated with a reduced risk of suicide and reduced repetition of attempted suicide. In addition, a healthy lifestyle, with maintenance of good diet and sleep habits, regular physical activity, abstinence from smoking and illicit drug use, is also associated with a reduced risk of suicidal behaviour.
                          In Tamilnadu adolencent health programme and Chief Minister.Jayalalitha’s computer schme to school students are very much helpful to prevent suicide among  students. Along with the easy  school education, better exam results in tamilnadu are also helpful to prevent suicide in students.
(ii)Respect of Self Esteem:
                        World Health Organization states that “Worldwide, suicide is among the top five causes of mortality in the 15-to 19-years age group and in many countries it makes first or second as a cause of death among both boys and girls in this age group”. And recommends “strengthening student’s self-esteem” to protect children and adolescents against mental distress and dependency, and enables them to cope adequately with difficult and stressful life situations. And “prevention bullying and violence at school” that specific skills should be available in the education system to prevent bullying and violence in and around the school promises in order create a safe environment free of intolerance. And as well “to de-stigmatize mental illness”.
Treatment:
                        There are various treatment modalities to reduce the risk of suicide by addressing the underlying conditions causing suicidal ideation, including, depending on case history, medical pharmacological and psychotherapeutic talk therapies.
                        The conservative estimate is that 10% of individuals with psychiatric disorders may have an undiagnosed medical condition causing their symptoms, upwards of 50% may have an undiagnosed medical condition which if not causing is exacerbating their psychiatric symptoms. Illegal drugs and prescribed medications may also produce psychiatric symptoms. Effective diagnosis and if necessary medical testing which mau include neuroimaging to diagnose and treat result of psychiatric symptoms, most often including depression, which are present in up to 90-95% of cases.
                        Recent research has shown that Lithium has been effective with lowering the risk of suicide in those with bipolar disorder to the same levels as the general population. Lithium has also proven effective in lowering the suicide risk in those with unipolar depression as well.   
                        There are multiple evidence-based psychotherapeutic talk therapies available to reduce suicidal ideation suth as dialectical behaviour therapy (DBT) for which multiple studies have reported varying degrees of clinical effectiveness in reducing suicidality. Benefits include a reduction in self-harm behaviours and suicidal ideations. Cognitive Behavior Therapy for suicide prevention (CBT-SP) is a form of DBT adapted for adolescents at high risk for repeated suicide attempts
Suicide prevention is possible:           
                        Suicide is a multi-determined phenomenon that occurs against a background of complex interacting biological, social, psychological and environmental rick and protective factors. Despite the complexity of this phenomenon, suicide can be prevented.
(i)Primary prevention:
                        Primary prevention of suicide requires broad modifications of social, economic and biological conditions to prevent members of a population from becoming suicidal. Primary prevention involves population-based interventions, rather than focusing on the individual at risk. Primary preventive interventions include restricting access to lethal methods, promoting physical health and positive mental health, promoting a responsible representation of suicide in social and other media, seeking to reduce stigmatization of mental illness and suicide and encouraging help-seeking behaviour through public awarness and education campaigns.
(ii)Secondary prevention:
                        Secondary prevention is aimed at minimising suicide risk in high-risk populations. In this sense, early identification of suicidal individuals, accurate diagnosis and effective treatment of mental health problems, especially mood disorders and substance-related disorders are crucial. More than half of the patients who die by suicide have seen their primary care physician within the month before their death. Therefore, improving primary care physicians’ recognition of psychiatric symptoms and disorders, suicide risk evaluation, treatment interventions and referral skills are key components of suicide prevention. Similarly, providing educational programs for “gatekeepers” (people who regularly come into contact with individuals or families in distress, such as clergy, first responders, pharmacists, teachers and police) can improve recognition of the risk of suicide and self-harm and facilitate referral of vulnerable people to appropriate assessment and treatment facilities.
(iii)Tertiary prevention:
                        Finally, tertiary prevention is aimed at preventing relapses of suicidal behaviour after a suicide attempt. This also involves the critical work of postvention __ the care, support and treatment of those impacted by suicide.
Suicide prevention in India:
                        In our country A three pronged attack to combat suicide are (1) reducing social isolation, (2) prevent social disintegration,and (3) treating mental disorders. Additionally a set of state led policies an being enforced to decrease the high suicide rate among farmors. Counselling program for school and college students along with their parents and teachers are also helpful to prevent suicide among students.                                                                                                                                        Suicides can be prevented through interventions like banning the most toxic pesticides and teaching rural communities on safe storage of pesticides. India should also start mental health promotion for toung people through schools and colleges and introduce crisis counseling services and services for treatment of depression and alcohol addiction.” In India current concern about suicides has focused on agricultural workers, over three in four suicide death in India occur in other occupational group( including those who are unemployed and homemakers).                                                                                                                                                                         Most suicide death occurred in rural areas –the age standardized death rates were about two times higher in rural than in urban areas. A reduced risk of suicide versus other causes of death in women who were widowed, divorced or separated, compared with married women and men. So marriage at appropriate age and time can also helpful to prevent suicide in reporductive age group.                                                                
                                                                                                        
 Dr.M. A.ALEEM.M.D.D.M.,(Neuro)                                                                           Consultant Neurologist and Epileptologist.                                                 Trained at Institution of neurology Queen’s Square London.                                                    Professor of Neurology KAPV Government Medical college.                                                     MGM Government Hospital Trichy -620017. Tamilnad,India.